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This proinflammatory signal induces epigenetic changes, promoting Th17 differentiation and expansion, enhancing resistance to intestinal infection but increasing risk of intestinal inflammation

Both of these processes caused the activation of inflammatory mediators via oxidative stress-induced redox imbalance

Rodier, Y

Thus, GSH supplementation in the DG sub-group of elderly diabetic patients over a period of 6 months led to a significant increase in the erythrocyte GSH, GSSG, and FPI and a decrease in HbA1c and 8-OHdG levels (Figure S2), suggesting that the elderly diabetic population responds better to GSH supplementation in conjunction with anti-diabetic therapy

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